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These ligands have the intrinsic ability to activate receptor signaling in the absence of an agonist while simultaneously performing the PAM effect [47, 247]
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It has also been observed to reduce the levels of paired helical filament (PHF), β-amyloid (βA) 1-40, and βA 1-42 and decreases the BACE1-mediated fragmentation of APP (shown in Figure 6) [373]
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