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LPS not only stimulates the phosphorylation of extracellular signal-regulated kinase (ERK1/2) and nuclear factor kappa-B (NF-κB), but also strongly increases the expression levels of IL-1β, IL-6, IL-12, VEGF, TNF, and TNF-related apoptosis-inducing ligand

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The Cancer Genome Atlas Expression subtypes stratify response to checkpoint inhibition in advanced urothelial cancer and identify a subset of patients with high survival probability