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The binding of Nrf2 to AREs is controlled by the small Maf proteins (MafG, MafK, and MafF), which form heterodimers with Nrf2, effective in the activation of AREs, but at the same time, Maf proteins can homodimerize with each other, preventing the activation of AREs by Nrf2 [193,194]
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Therefore, in the present study, we used 100 µg/mL GSH to study the anti-inflammatory effect and underlying mechanism
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